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Daniel J. Drucker, Jeffrey M. Friedman, Joel F. Habener, Jens Juul Holst and Svetlana Mojsov 2024 Princess of Asturias Award for Technical & Scientific Research

Daniel J. Drucker, Jeffrey M. Friedman, Joel F. Habener, Jens Juul Holst and Svetlana Mojsov

In recent years, a great deal of progress has been made in the treatment of type 2 diabetes, with the appearance of drugs that use semaglutide as an active ingredient, a peptide similar to a hormone, glucagon-like peptide 1 (GLP-1), which plays a counterbalancing role for insulin in blood sugar balance. When the sugar level drops, glucagon induces the liver to release glucose, and when it rises, more insulin is generated, which is responsible for reducing the excess. Furthermore, semaglutide produces a notable reduction in appetite, which has significantly contributed to the success of Ozempic, one of the drugs produced with this active ingredient. Science magazine (Prince of Asturias Award for Communication and Humanities 2007) designated these anti-obesity drugs as the greatest scientific advance of 2023. Drucker, Habener, Holst and Mojsov share the recognition of having initiated and developed this research since the 1970s. At their different laboratories, they have studied the hormones that intervene in the process and regulate digestive metabolism, such as somatostatin, which inhibits the production of glucagon and insulin, and variants of glucagon, called GLP-1 and GLP-2, and verified that this homeostasis system could be an effective therapeutic target against type-2 diabetes. Semaglutide (like some other similar molecules) works as an agonist of the GLP-1 receptor, thereby inhibiting the production of this hormone, reducing blood sugar levels and improving the growth of pancreatic beta (β)-cells, responsible for the production and release of insulin. In addition, it has been proven to have protective effects against vascular accidents in adults with obesity, an indication recently authorized by the U.S. Food and Drug Administration (FDA). For his part, Friedman discovered another hormone in 1994, leptin, which is generated in fat cells, or adipocytes, and acts on the brain region that controls appetite. It is a balanced system: the more fat there is, the more leptin is produced, which decreases appetite, thus reducing the body’s fat and hence the production of leptin. In the case of obese people, this mechanism is unbalanced. He has also studied the genetic predisposition to obesity.

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